论文标题
关键性如何达到无序固体压缩故障的分叉
How criticality meets bifurcation in compressive failure of disordered solids
论文作者
论文摘要
Continuum Mechanics将压缩故障描述为材料对增加负载的响应的标准分叉:损伤最初在材料中均匀生长,将其定位在薄带中。然而,在定位证据幂律分布式故障前体之前记录了大小增加的实验活性的实验,这表明压缩故障是关键现象。我们在这里检查了这种明显的矛盾,通过探测损伤活动的空间组织及其演变,直到在2D细胞固体的压缩实验中定位为止。我们实验中测得的间歇性损害进化被非平稳的静态方程式充分描述,这些方程是由损害力学和让人联想到关键现象的。在此描述中,前体是造成损害的损害,从材料的障碍与个人损害事件后的远距离应力再分配之间产生了损害。然而,我们在实验中观察到的几乎失败的特征大小的差异并不是向关键性过渡的签名。取而代之的是,在伤害演变的所有阶段,系统保持固定距离到临界点。差异是由于材料驱动到定位时的稳定性逐渐丧失而导致的。因此,我们的研究表明,压缩故障是材料障碍起着边缘作用的标准分叉。它还表明,前保期活动构成了对定位的进化的副产品,并且可以建立一种预测方法来评估结构的剩余寿命。
Continuum mechanics describes compressive failure as a standard bifurcation in the response of a material to an increasing load: damage, which initially grows uniformly in the material, localizes within a thin band at failure. Yet, experiments recording the acoustic activity preceding localization evidence power-law distributed failure precursors of increasing size, suggesting that compressive failure is a critical phenomenon. We examine here this apparent contradiction by probing the spatial organization of the damage activity and its evolution until localization during compression experiments of 2D cellular solids. The intermittent damage evolution measured in our experiments is adequately described by a non-stationary depinning equation derived from damage mechanics and reminiscent of critical phenomena. In this description, precursors are damage cascades emerging from the interplay between the material's disorder and the long-range stress redistributions following individual damage events. Yet, the divergence of their characteristic size close to failure, which we observe in our experiments, is not the signature of a transition towards criticality. Instead, the system remains at a fixed distance to the critical point at all stages of the damage evolution. The divergence results from the progressive loss of stability of the material as it is driven towards localization. Thus, our study shows that compressive failure is a standard bifurcation for which the material disorder plays a marginal role. It also shows that precursory activity constitute by-products of the evolution towards localization and can serve to build a predictive method to assess the residual lifetime of structures.