论文标题
注射后无性抗震动是一种延迟触发地震性的机制
Post-injection aseismic slip as a mechanism for the delayed triggering of seismicity
论文作者
论文摘要
从剥削地球资源到对地震的理解,注射诱导的无性滑移在广泛的人制和自然系统中起着重要作用。最近的研究揭示了Aseassic滑移如何响应连续的流体注射。然而,对注射流体后断层的响应已停止,知之甚少。在这项工作中,我们通过水力力学模型调查了所谓的注射后阶段的繁殖和最终停滞。我们表明,关闭后,故障滑移以脉冲状模式传播。控制传播作为脉搏的条件,尤其是何时何时何时何地破裂停滞。尤其是,严重的压力断层可以容纳破裂的脉冲,这些断裂脉冲在注射持续时间的几个数量级传播,并且在关闭时的破裂尺寸几乎是破裂的大小的两倍。因此,我们认为,持续的压力越来越大,越来越大的岩石量是由注射后的抗震动滑移引起的,这是触发注射后地震性的合理机制,这是地球能源行业的关键问题。我们讨论了基于记录的注射后诱导的地震性病例来支持这种机制的证据。
Injection-induced aseismic slip plays an important role in a broad range of human-made and natural systems, from the exploitation of geo-resources to the understanding of earthquakes. Recent studies have shed light on how aseismic slip propagates in response to continuous fluid injections. Yet much less is known about the response of faults after the injection of fluids has stopped. In this work, we investigate via an hydro-mechanical model the propagation and ultimate arrest of aseismic slip during the so-called post-injection stage. We show that after shut-in, fault slip propagates in pulse-like mode. The conditions that control the propagation as a pulse and notably when and where the ruptures arrest are fully established. In particular, critically-stressed faults can host rupture pulses that propagate for several orders of magnitude the injection duration and reach up to nearly double the size of the ruptures at the moment of shut-in. We consequently argue that the persistent stressing of increasingly larger rock volumes caused by post-injection aseismic slip is a plausible mechanism for the triggering of post-injection seismicity -- a critical issue in the geo-energy industry. We discuss evidence that supports this mechanism based on documented cases of post-injection-induced seismicity.